Author Technische Universitaet Muenchen

Image by Konnerth lab, TU Muenchen
Single-neuron observations mark steps in Alzheimer’s disease

Studying a mouse model of Alzheimer’s disease, neuroscientists at the Technische Universitaet Muenchen have observed correlations between increases in both soluble and plaque-forming beta-amyloid – a protein implicated in the disease process – and dysfunctional developments on several levels: individual cortical neurons, neuronal circuits, sensory cognition, and behavior.