Alcohol

Early results from a pilot study indicate that psilocybin-assisted therapy could be linked to lower alcohol consumption and improved psychological outcomes, though larger controlled trials are needed to determine whether the psilocybin itself is responsible for these changes.

A new study suggests that people who feel fewer bodily sensations while intoxicated may be at higher risk for alcohol misuse. Using bodily maps and heartbeat tasks, researchers linked internal awareness to physiological effects and subjective feelings of intoxication.

A new study of heavy-drinking young adults found that drinking more on a given day was linked to both regretted and positive romantic or sexual experiences. For women, using cannabis alongside alcohol appeared to reduce the likelihood of regret.

The length of your fingers might predict how much alcohol you drink, according to a new study linking prenatal hormone exposure to alcohol use in university students.

A small clinical trial finds semaglutide may help people with alcohol use disorder cut back on drinking and reduce cravings.

Drinking alcohol in social settings is often more enjoyable, and new research explains why: the positive effects of alcohol are actually amplified when your conversation partner is also under the influence.

A new study in PNAS Nexus reveals that communities experiencing mass shootings see sustained increases in alcohol sales.

A study found that individuals with alcohol use disorder show reduced gray matter in cortical and limbic areas. These brain changes correlate with increased anxiety, depression, and memory deficits.

New research highlights a potential mechanism behind intoxicated aggression.

The use of diabetes drugs semaglutide and liraglutide was associated with fewer hospitalizations for alcohol use disorder compared to approved treatments, suggesting these medications may have potential benefits for managing alcohol-related health issues.

Researchers found increased thalamus-insula connectivity in individuals with alcohol use disorder during unpredictable threats, linked to higher alcohol intake. This suggests heightened threat reactivity may drive excessive drinking.

A new study has uncovered a critical molecular mechanism behind alcohol's effects on the brain. Researchers found that a previously uncharacterized protein, TMEM132B, amplifies alcohol's influence on GABAA receptors.

Alzheimer’s disease and alcohol use disorder share similar gene expression changes, including increased inflammation, disrupted brain signaling, and vascular damage, suggesting alcohol use may worsen or accelerate Alzheimer’s progression through shared molecular mechanisms.