Scientists have used high resolution imaging to discover that brain networks we use to think about others are connected to specific parts of the amygdala, an ancient region deep inside the brain.
Researchers used genetic data from over a million people and brain scans from over 30,000 to link genetic predisposition to dyslexia with differences in brain structure, particularly in areas involved in motor skills, vision, and language.
Recent research found that healthy aging is associated with widespread brain network changes, some linked to preserved thinking skills. In contrast, semantic dementia is associated with more rigid brain connections, particularly in the temporal and parietal regions, along with cognitive...
A large-scale neuroimaging study found no evidence of atypical functional connectivity in the amygdala of individuals with autism, challenging previous hypotheses. Results suggest amygdala connectivity differences may not be a core feature of autism.
Researchers discovered that amyloid precursor protein interacts with talin to maintain synaptic stability. Disruptions in this interaction may lead to Alzheimer’s disease by impairing mechanical signaling, triggering synaptic degeneration, and promoting amyloid plaque formation.
Men with psychiatric symptoms and more severe anhedonia showed increased functional connectivity between the paraventricular nucleus of the thalamus and the nucleus accumbens, suggesting a neural basis for anhedonia linked to reward-processing brain regions.
New neurons in adult brains are linked to improved verbal learning and reduced cognitive decline, highlighting their unique role in humans compared to mice and their potential as a target for future cognitive-enhancing treatments.
Childhood maltreatment alone showed no effect on hippocampal size, but individuals with maltreatment and a romantic breakup had smaller hippocampal volumes, highlighting how later-life stress can exacerbate early trauma's impact on brain structure.
ADHD stems from neurodevelopmental differences, including altered brainwaves, smaller brain regions, reduced blood flow, and lower dopamine levels. These insights highlight ADHD’s neurological roots, shifting diagnosis from behavior-based to brain-focused approaches.
Neuroinflammation, caused by infections or chronic diseases, depletes muscle energy via brain-to-muscle cytokine signaling, leading to fatigue without damaging muscle fibers. Targeting this pathway with treatments like neutralizing antibodies shows promise for improving muscle function.
Norepinephrine-driven oscillations during non-REM sleep power the brain's glymphatic system to clear toxic proteins, but the sleep aid zolpidem disrupts this process, impairing waste clearance and potentially increasing neurodegenerative disease risk.
A meta-analysis of 38 trials found neurofeedback has limited benefits for ADHD, showing small improvements in processing speed with standard protocols but no meaningful impact on core symptoms.
Researchers observed that reward system activation differed in individuals with PTSD, with variations linked to depressive symptoms, resilience, and trauma exposure.
Smoldering eucalyptus wood smoke extract increases inflammation and disrupts the blood-brain barrier by reducing tight junction proteins, suggesting wildfire smoke may contribute to neuroinflammation and elevate the risk of neurological disorders.
A study found that levodopa-induced dyskinesia disconnects the motor cortex from movement control, allowing abnormal movements. Ketamine reduced these movements, restored some brain control, and altered neural interactions, showing promise as a potential treatment.